Bcl-2 anticorps (AA 41-54)
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- Antigène Voir toutes Bcl-2 (BCL2) Anticorps
- Bcl-2 (BCL2) (B-Cell CLL/lymphoma 2 (BCL2))
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Épitope
- AA 41-54
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Reactivité
- Humain
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Hôte
- Souris
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Clonalité
- Monoclonal
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Conjugué
- Cet anticorp Bcl-2 est non-conjugé
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Application
- Western Blotting (WB), Immunohistochemistry (IHC), ELISA, Flow Cytometry (FACS), Immunofluorescence (IF), Immunoprecipitation (IP)
- Séquence
- GAAPAPGIFS SQPG
- Réactivité croisée
- Humain, Singe, Porc
- Réactivité croisée (Details)
- Calculated cross reactivity: Hu Mk Po
- Attributs du produit
- Bcl-2 (Apoptosis Regulator Bcl-2, B Cell CLL/lymphoma-2)
- Purification
- Purified by Protein A/G affinity chromatography.
- Immunogène
- Synthetic peptide corresponding to aa41-54 (GAAPAPGIFSSQPG-Cys) from human Bcl-2.
- Clone
- 8C8
- Isotype
- IgG1 kappa
- Top Product
- Discover our top product BCL2 Anticorps primaire
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- Indications d'application
- Optimal working conditions should be determined by the investigator.
- Restrictions
- For Research Use only
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- Format
- Liquid
- Buffer
- Supplied as a liquid in PBS, 0.05 % BSA, 0.05 % sodium azide.
- Agent conservateur
- Sodium azide
- Précaution d'utilisation
- This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
- Stock
- -20 °C
- Stockage commentaire
- -20°C
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- Antigène
- Bcl-2 (BCL2) (B-Cell CLL/lymphoma 2 (BCL2))
- Autre désignation
- Bcl-2 (BCL2 Produits)
- Synonymes
- anticorps Bcl-2, anticorps PPP1R50, anticorps bcl-2, anticorps AW986256, anticorps C430015F12Rik, anticorps D630044D05Rik, anticorps D830018M01Rik, anticorps BCL-2, anticorps Bcl2, anticorps BCL2, apoptosis regulator, anticorps B cell leukemia/lymphoma 2, anticorps B-cell CLL/lymphoma 2a, anticorps B-cell CLL/lymphoma 2 protein, anticorps BCL2, anticorps Bcl2, anticorps bcl2a, anticorps BCL-2
- ID gène
- 150749
- NCBI Accession
- NP_000624
- UniProt
- P10415
- Pathways
- Signalisation MAPK, Signalisation PI3K-Akt, Apoptose, Caspase Cascade in Apoptosis, Regulation of Muscle Cell Differentiation, Cell-Cell Junction Organization, Skeletal Muscle Fiber Development, Autophagy, Smooth Muscle Cell Migration, Negative Regulation of intrinsic apoptotic Signaling
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