MMAB anticorps
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- Antigène Voir toutes MMAB Anticorps
- MMAB (Methylmalonic Aciduria (Cobalamin Deficiency) CblB Type (MMAB))
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Reactivité
- Humain
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Hôte
- Souris
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Clonalité
- Monoclonal
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Conjugué
- Cet anticorp MMAB est non-conjugé
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Application
- Western Blotting (WB), Flow Cytometry (FACS), Immunofluorescence (IF)
- Attributs du produit
- Homo sapiens methylmalonic aciduria (cobalamin deficiency) cblB type (MMAB), nuclear gene encoding mitochondrial protein, transcript variant 1
- Purification
- Purified from mouse ascites fluids by affinity chromatography
- Immunogène
- Full length human recombinant protein of human MMAB (NP_443077) produced in HEK293T cell.
- Clone
- 3B8
- Isotype
- IgG2a
- Top Product
- Discover our top product MMAB Anticorps primaire
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- Indications d'application
- WB 1:2000, IF 1:100, FLOW 1:100
- Commentaires
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The concentration of the product may vary between diferrent lots.
- Restrictions
- For Research Use only
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- Format
- Liquid
- Concentration
- 0.5-1.0 mg/mL
- Buffer
- PBS (PH 7.3) containing 1 % BSA, 50 % glycerol and 0.02 % sodium azide.
- Agent conservateur
- Sodium azide
- Précaution d'utilisation
- This product contains sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
- Stock
- -20 °C
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- Antigène
- MMAB (Methylmalonic Aciduria (Cobalamin Deficiency) CblB Type (MMAB))
- Autre désignation
- MMAB (MMAB Produits)
- Synonymes
- anticorps ATR, anticorps cblB, anticorps cob, anticorps 9130222L19Rik, anticorps methylmalonic aciduria (cobalamin deficiency) cblB type, anticorps methylmalonic aciduria (cobalamin deficiency) cblB type homolog (human), anticorps MMAB, anticorps Mmab
- Sujet
- This gene encodes a protein that catalyzes the final step in the conversion of vitamin B(12) into adenosylcobalamin (AdoCbl), a vitamin B12-containing coenzyme for methylmalonyl-CoA mutase. Mutations in the gene are the cause of vitamin B12-dependent methylmalonic aciduria linked to the cblB complementation group.
- Poids moléculaire
- 24 kDa
- ID gène
- 326625
- NCBI Accession
- NM_052845
- HGNC
- 326625
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