ATP6V0D2 anticorps (Middle Region)
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- Antigène Voir toutes ATP6V0D2 Anticorps
- ATP6V0D2 (ATPase, H+ Transporting, Lysosomal 38kDa, V0 Subunit D2 (ATP6V0D2))
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Épitope
- Middle Region
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Reactivité
- Humain, Souris, Rat, Lapin, Chien, Cobaye, Boeuf (Vache), Cheval, Porc
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Hôte
- Lapin
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Clonalité
- Polyclonal
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Conjugué
- Cet anticorp ATP6V0D2 est non-conjugé
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Application
- Western Blotting (WB)
- Séquence
- GLRLLAQAED FDQMKNVADH YGVYKPLFEA VGGSGGKTLE DVFYEREVQM
- Homologie
- Cow: 100%, Dog: 100%, Guinea Pig: 86%, Horse: 93%, Human: 100%, Mouse: 100%, Pig: 100%, Rabbit: 100%, Rat: 100%
- Attributs du produit
- This is a rabbit polyclonal antibody against ATP6V0D2. It was validated on Western Blot using a cell lysate as a positive control.
- Purification
- Affinity Purified
- Immunogène
- The immunogen is a synthetic peptide directed towards the middle region of human ATP6V0D2
- Top Product
- Discover our top product ATP6V0D2 Anticorps primaire
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- Indications d'application
- Optimal working dilutions should be determined experimentally by the investigator.
- Commentaires
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Antigen size: 350 AA
- Restrictions
- For Research Use only
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- Format
- Liquid
- Concentration
- Lot specific
- Buffer
- Liquid. Purified antibody supplied in 1x PBS buffer with 0.09 % (w/v) sodium azide and 2 % sucrose.
- Agent conservateur
- Sodium azide
- Précaution d'utilisation
- This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
- Conseil sur la manipulation
- Avoid repeated freeze-thaw cycles.
- Stock
- -20 °C
- Stockage commentaire
- For short term use, store at 2-8°C up to 1 week. For long term storage, store at -20°C in small aliquots to prevent freeze-thaw cycles.
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Predicting and validating the pathway of Wnt3a-driven suppression of osteoclastogenesis." dans: Cellular signalling, Vol. 26, Issue 11, pp. 2358-69, (2014) (PubMed).
: "Low-density lipoprotein receptor deficiency causes impaired osteoclastogenesis and increased bone mass in mice because of defect in osteoclastic cell-cell fusion." dans: The Journal of biological chemistry, Vol. 287, Issue 23, pp. 19229-41, (2012) (PubMed).
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Predicting and validating the pathway of Wnt3a-driven suppression of osteoclastogenesis." dans: Cellular signalling, Vol. 26, Issue 11, pp. 2358-69, (2014) (PubMed).
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- Antigène
- ATP6V0D2 (ATPase, H+ Transporting, Lysosomal 38kDa, V0 Subunit D2 (ATP6V0D2))
- Autre désignation
- ATP6V0D2 (ATP6V0D2 Produits)
- Synonymes
- anticorps ATP6D2, anticorps VMA6, anticorps AI324824, anticorps V-ATPase, anticorps 1620401A02Rik, anticorps ATPase H+ transporting V0 subunit d2, anticorps ATPase, H+ transporting, lysosomal V0 subunit D2, anticorps ATPase H+ transporting V0 subunit D2, anticorps ATPase, H+ transporting, lysosomal 38kDa, V0 subunit d2 L homeolog, anticorps ATP6V0D2, anticorps Atp6v0d2, anticorps atp6v0d2.L
- Sujet
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ATP6V0D2 is the subunit of the integral membrane V0 complex of vacuolar ATPase. Vacuolar ATPase is responsible for acidifying a variety of intracellular compartments in eukaryotic cells, thus providing most of the energy required for transport processes in the vacuolar system. ATP6V0D2 may play a role in coupling of proton transport and ATP hydrolysis.
Alias Symbols: ATP6D2, FLJ38708, VMA6
Protein Interaction Partner: AGO3, RBM15B, RPS3, NDUFB8, ADRM1,
Protein Size: 350 - Poids moléculaire
- 40 kDa
- ID gène
- 245972
- NCBI Accession
- NM_152565, NP_689778
- UniProt
- Q8N8Y2
- Pathways
- Transition Metal Ion Homeostasis, Proton Transport
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