BCR anticorps (Peroxidase (POD))
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- Antigène Voir toutes BCR Anticorps
- BCR (Breakpoint Cluster Region (BCR))
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Reactivité
- Toutes les espèces
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Hôte
- Souris
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Clonalité
- Monoclonal
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Conjugué
- Cet anticorp BCR est conjugé à/à la Peroxidase (POD)
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Application
- Immunohistochemistry (IHC)
- Purification
- The splenic lymphocytes from BALB/c mouse, immunized with CML-HSA were fused tomyeloma P3U1 cells. The cell line (NF-1G) with positive reaction was grown in ascitic fluid ofBALB/c mouse, from which the antibody was purified by Protein G affinity chromatography,and conjugated
- Immunogène
- CML-HSA
- Clone
- NF-1G
- Isotype
- IgG2a
- Top Product
- Discover our top product BCR Anticorps primaire
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- Indications d'application
- Working dilution for immunohistochemistry: 3-10ug/ml,
- Restrictions
- For Research Use only
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- Concentration
- 0.25 mg/ml
- Buffer
- PBS (containing 2% Block Ace as a stabilizer, 0.1% Proclin as a bacteriostat)
- Agent conservateur
- ProClin
- Stock
- -20 °C
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- Antigène
- BCR (Breakpoint Cluster Region (BCR))
- Autre désignation
- CML (BCR Produits)
- Synonymes
- anticorps si:dkey-83d9.7, anticorps zK83D9.7, anticorps BCR, anticorps ALL, anticorps BCR1, anticorps CML, anticorps D22S11, anticorps D22S662, anticorps PHL, anticorps 5133400C09Rik, anticorps AI561783, anticorps AI853148, anticorps mKIAA3017, anticorps breakpoint cluster region, anticorps BCR, RhoGEF and GTPase activating protein, anticorps bcr, anticorps BCR, anticorps Bcr
- Sujet
- Reaction of protein amino groups with glucose leads, through the early products such as a Schiff base andAmadori rearrangement products, to the formation of advanced glycation end products (AGEs). Recentimmunological studies using anti-AGEs antibody (6D12) demonstrated the presence of AGEs-modified proteinsin several human tissues: human lens (nondiabetic and noncataractous), renal proximal tubules inpatients with diabetic nephropathy and chronic renal failure, diabetic retina, peripheral nerves ofdiabetic neuropathy, atherosclerotic lesions of arterial walls, beta2-microglobulin forming amyloid fibrilsin patients with hemodialysis-related amyloidosis, senile plaques of patients with Alzheimer
- Pathways
- Regulation of Leukocyte Mediated Immunity, Platelet-derived growth Factor Receptor Signaling
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