Bax Inhibitor 1 anticorps

N° du produit ABIN347215

Détail du produit anti-Bax Inhibitor 1 anticorps

Antigène: Bax Inhibitor 1 (TMBIM6) (Transmembrane BAX Inhibitor Motif Containing 6 (TMBIM6))

Reactivité: Oryza sativa

Hôte: Lapin

Clonalité: Polyclonal

Conjugué: Cet anticorp Bax Inhibitor 1 est non-conjugé

Application: Western Blotting (WB), ELISA

Specificité: Reacts with Oryza sativa subsp

Réactivité croisée (Details): japonica (Rice) 27 kDa BI-1 protein.

Purification: Antiserum

Immunogène: Synthetic peptide derived from Bax inhibitor-1 protein of Oryza sativa subsp. Japonica (Rice)

Isotype: IgG

Information d'application

Indications d'application:

Working dilution: Optimal dilution should be determined by the end user.
The following are guidelines only :
WB(1:500 - 1:5 000)

Restrictions: For Research Use only

Stockage

Format: Lyophilized

Reconstitution: Must be reconstituted in distilled water.

Stock: 4 °C/-20 °C

Stockage commentaire: Lyophilized powder stable for a minimum of 2 years at -20°C. Store reconstituted antibodies at +4°C. For extended periods store in aliquots at -20°C. Antibodies are guaranteed for 6 month from date of receipt.

Date de péremption: 24 months

Détails sur Bax Inhibitor 1

Antigène: Bax Inhibitor 1 (TMBIM6) (Transmembrane BAX Inhibitor Motif Containing 6 (TMBIM6))

Autre désignation: Bax Inhibitor-1 (TMBIM6 Produits)

Synonymes: anticorps ARABIDOPSIS BAX INHIBITOR 1, anticorps ATBI-1, anticorps ATBI1, anticorps BAX INHIBITOR 1, anticorps BAX inhibitor 1, anticorps BI-1, anticorps K14A3.7, anticorps K14A3_7, anticorps BAXI1, anticorps TEGT, anticorps 5031406P05Rik, anticorps Bi1, anticorps Tegt, anticorps Ab1-011, anticorps Ac1-149, anticorps Cc1-27, anticorps BAX inhibitor 1, anticorps transmembrane BAX inhibitor motif containing 6, anticorps BI1, anticorps TMBIM6, anticorps Tmbim6

Sujet: Accelerates programmed cell death by binding to, and antagonizing the apoptosis repressor BCL2 or its adenovirus homolog E1B 19k protein. Under stress conditions, undergoes a conformation change that causes translocation to the mitochondrion membrane, leading to the release of cytochrome c that then triggers apoptosis. Promotes activation of CASP3, and thereby apoptosis

UniProt: Q9MBD8

Pathways: Response to Water Deprivation, ER-Nucleus Signaling