BAZ1B anticorps (N-Term)

N° du produit ABIN965640

Détail du produit anti-BAZ1B anticorps

Antigène: BAZ1B (Bromodomain Adjacent To Zinc Finger Domain, 1B (BAZ1B))

Épitope: N-Term

Reactivité: Humain

Hôte: Lapin

Clonalité: Polyclonal

Conjugué: Cet anticorp BAZ1B est non-conjugé

Application: Immunohistochemistry (IHC)

Purification: Purified by antigen-specific affinity chromatography.

Immunogène: Polyclonal antibody produced in rabbits immunizing with a synthetic peptide corresponding to N-terminal residues of human BAZ1B (bromodomain adjacent to zinc finger domain, 1B)

Information d'application

Indications d'application: ELISA, Western blotting: 1µg/ml for 2hrs.

Restrictions: For Research Use only

Stockage

Format: Liquid

Buffer: This antibody is stored in PBS, 50% glycerol

Agent conservateur: Sodium azide

Précaution d'utilisation: This product contains sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.

Stock: -20 °C

Références pour anticorps anti-BAZ1B (ABIN965640)

Lu, Meng, Morris, Keating: "A novel human gene, WSTF, is deleted in Williams syndrome." dans: Genomics, Vol. 54, Issue 2, pp. 241-9, (1999) (PubMed).

Détails sur BAZ1B

Antigène: BAZ1B (Bromodomain Adjacent To Zinc Finger Domain, 1B (BAZ1B))

Autre désignation: BAZ1B (BAZ1B Produits)

Synonymes: anticorps BAZ1B, anticorps wstf, anticorps wbscr9, anticorps wbscr10, anticorps LOC100220420, anticorps WBSCR10, anticorps WBSCR9, anticorps WSTF, anticorps C87820, anticorps Wbscr9, anticorps fi60d02, anticorps im:7137554, anticorps wu:fi60d02, anticorps bromodomain adjacent to zinc finger domain 1B, anticorps bromodomain adjacent to zinc finger domain, 1B, anticorps bromodomain adjacent to zinc finger domain 1B S homeolog, anticorps BAZ1B, anticorps baz1b, anticorps Baz1b, anticorps baz1b.S

Sujet: The BAZ1B (bromodomain adjacent to zinc finger domain, 1B) is a member of the bromodomain protein family. The bromodomain is a structural motif characteristic of proteins involved in chromatin-dependent regulation of transcription. This gene is deleted in Williams-Beuren syndrome, a developmental disorder caused by deletion of multiple genes at 7q11.23. Alternative splicing of this gene generates at least 2 transcript variants.

Pathways: Nuclear Hormone Receptor Binding, Chromatin Binding