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Daf-2 anticorps (C-Term)

DAF2 Reactivité: C. elegans IHC Hôte: Lapin Polyclonal unconjugated
N° du produit ABIN965970
  • Antigène Tous les produits Daf-2 (DAF2)
    Daf-2 (DAF2) (Decay Accelerating Factor 2 (DAF2))
    Épitope
    C-Term
    Reactivité
    C. elegans
    Hôte
    Lapin
    Clonalité
    Polyclonal
    Application
    Immunohistochemistry (IHC)
    Purification
    Purified by antigen-specific affinity chromatography.
    Immunogène
    Polyclonal antibody produced in rabbits immunizing with a synthetic peptide corresponding to C-terminal residues of Caenorhabditis elegans daf-2(Abnormal dauer formation protein 2)
  • Indications d'application
    ELISA, Western blotting: 1µg/ml for 2hrs.
    Restrictions
    For Research Use only
  • Format
    Liquid
    Buffer
    This antibody is stored in PBS, 50% glycerol
    Agent conservateur
    Sodium azide
    Précaution d'utilisation
    This product contains sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
    Stock
    -20 °C
  • Antigène
    Daf-2 (DAF2) (Decay Accelerating Factor 2 (DAF2))
    Autre désignation
    Daf-2
    Synonymes
    anticorps AI323748, anticorps Cd55b, anticorps Daf, anticorps Daf-TM, anticorps TM-DAF, anticorps CD55 molecule, decay accelerating factor for complement B, anticorps Insulin-like receptor subunit beta, anticorps Cd55b, anticorps daf-2
    Sujet
    The daf-2 is an insulin receptor-like gene that regulates longevity and diapause in Caenorhabditis elegans. A C. elegans neurosecretory signaling system regulates whether animals enter the reproductive life cycle or arrest development at the long-lived dauer diapause stage. daf-2, a key gene in the genetic pathway that mediates this endocrine signaling, encodes an insulin receptor family member. Decreases in DAF-2 signaling induce metabolic and developmental changes, as in mammalian metabolic control by the insulin receptor. Decreased DAF-2 signaling also causes an increase in life-span. Lifespan regulation by insulin-like metabolic control is analogous to mammalian longevity enhancement induced by caloric restriction, suggesting a general link between metabolism, diapause, and longevity.
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